GUT MICROBIOTA AS A REGULATOR OF PHYSIOLOGICAL HOMEOSTASIS: MECHANISMS AND SYSTEMIC INTERACTIONS
DOI:
https://doi.org/10.37547/Keywords:
gut microbiota, physiological homeostasis, dysbiosis, metabolic balance, immune system, short-chain fatty acids, gut–brain axis, microorganisms, neuroendocrine system, systemic interactions, inflammatory processes, metabolic diseases, microbiota modulation, health and disease.Abstract
This work is devoted to the study of the important role of gut microbiota in the regulation of physiological homeostasis. The community of microorganisms inhabiting the human intestine actively participates in maintaining metabolic balance, regulating immune system activity, and influencing neuroendocrine processes. The study analyzes the mechanisms by which microbiota affects the host organism, including the production of metabolites such as short-chain fatty acids, modulation of the immune response, and interactions through the gut–brain axis.
Downloads
References
1.“General Physiology” – Axmedov, Sh. (2017). Tashkent: Publishing House of the Uzbekistan State Medical University.
2.“Human Physiology” – Toshpulatov, A., & Sobirov, D. (2019). Tashkent: Medical Publishing House.
3.“Microbiology” – Abdukarimov, S. (2018). Tashkent: Publishing House of the Uzbekistan State Medical University.
4.“Immunology” – Rahimov, A., & Sobirov, D. (2020). Tashkent: TDTU Publishing House.
5.“Digestive System and Microbiota” – Niyozov, R. (2019). Tashkent: TDTU Publishing House.
6.“Fundamentals of Biotechnology” – Karimov, F. (2019). Tashkent: Fan va Texnologiya Publishing House.
Downloads
Published
Issue
Section
License
This work is licensed under a Creative Commons Attribution 4.0 International License.
Articles published in this journal are licensed under the Creative Commons Attribution 4.0 International License (CC-BY 4.0). Under this license:
- Share: Copy and redistribute the material in any medium or format
- Adapt: Remix, transform, and build upon the material for any purpose, including commercially
Attribution required: You must give appropriate credit, provide a link to the license, and indicate if changes were made.
License URL: https://creativecommons.org/licenses/by/4.0/
Authors retain copyright of their work while granting the journal first publication rights.
